Professor Bryan Sykes: “Yeti hairs genetically identical to polar bear”
Research by a British scientist has concluded that the legendary Himalayan yeti may in fact be a sub-species of brown bear.
DNA tests on hair samples carried out by Oxford University genetics professor Bryan Sykes found that they matched those from an ancient polar bear.
He subjected the hairs to the most advanced tests available.
He says the most likely explanation for the myth is that the animal is a hybrid of polar bears and brown bears.
Prof Sykes told the BBC that there may be a real biological animal behind the yeti myth.
“I think this bear, which nobody has seen alive,… may still be there and may have quite a lot of polar bear in it,” he said.
“It may be some sort of hybrid and if its behaviour is different from normal bears, which is what eyewitnesses report, then I think that may well be the source of the mystery and the source of the legend.
DNA samples and video footage of an alleged Bigfoot sleeping in the Kentucky woods have been presented this week by researchers in Texas who say it all belongs to a ‘human hybrid.’
Bigfoot is real, and there’s now both DNA and video evidence to prove it, claims one group of devoted Sasquatch researchers.
The group’s “never-before-seen footage” of an alleged Bigfoot creature sleeping in the woods of Kentucky has been presented this week along with various blood and hair samples said to be unlike anything seen before.
The group’s startling statements are supposedly backed by 11 outside laboratories and universities, which all reviewed the findings, and which were provided with blind samples, according to the report by the Sasquatch Genome Project.
“We want people to understand that this is a serious study,” Dr. Melba Ketchum, a genetics scientist, who led the project during the course of the five-year study, told CBS DFW.
Unsurprisingly, others are challenging Ketchum’s credibility, including New York University whose laboratory Ketchum claims similarly tested a field sample and found it having usual human mitochondrial results.
In 2001, scientists announced an amazing discovery: the oldest skull of a human ancestor ever found. The 3½ million year old fossil was remarkably complete, and unlike any previous fossil find. Its discovery – by a team led by Meave Leakey of the famous Leakey fossil-hunting family – has revolutionised our understanding of how humans evolved.
The great mystery of our evolution is how an ape could have evolved into the extraordinary creature that is a human being. There has never been another animal like us on the planet. And yet ten million years ago there was no sign that humans would take over the world. Instead the Earth was dominated by the apes. More than 50 different species of ape roamed the world – ten million years ago Earth really was the planet of the apes. Three million years later, most had vanished. In their place came something clearly related to the apes, but also completely different: human beings!
Big pharma can’t allow cancer or any other disease to be cured because they will lose their business of drugs, pills and all the illness causing products they make and distribute to the dis-eased. Plus the elite need people to be sick and die to depopulate the world. Curing cancer has no benefit to them whatsoever even though the cure has been made and known for decades. Medicinal herbs and other forms of plants are known to be beneficial in eliminating cancer.
In an effort to facilitate discussion about genomics, the Center for Individualized Medicine at Mayo Clinic recently released 10 reasons why the genome matters for diagnosis and treatment.
It’s already influenced drug labels and uses for some medicines. The U.S. Food and Drug Administration has changed medication labeling, doses and uses based on genomic research, including a key drug for breast cancer.
It’s making sure your doctor is giving you the right drug at the right dose. Not everyone can tolerate or metabolize the same drugs; genomics shows some medications have no effect or might hurt rather than help certain individuals. For example, one person in ten cannot tolerate the standard treatment for irritable bowel disease and needs a different drug.
It makes developing new medical tests faster and better. It has already resulted in several new tests to diagnose and treat disease, and to avoid some needless therapies, including a treatment test for mood disorders.
It may be helping you prevent an illness. It can tell you that you have a greater susceptibility to some conditions, so you can change your behavior to avoid them. For example, those at risk for developing diabetes can make lifestyle changes to lower their risk.
It may be helping you or a family member fight a treatment-resistant disease. A genomic scan may reveal a genetic alteration that indicates an alternative, successful treatment. In one case, a mutation led to use of a kidney
Scientists have sequenced the genome of Botryllus schlosseri – a small sea creature which is humans’ closest living invertebrate relative.
The advance will make it possible to find the genetic basis for some of the animal’s amazing regenerative abilities and immunity features, which potentially could be applied to human medicine.
Botryllus schlosseri fuses together with others to form colonies that look like psychedelic blobs, encrusting rocks and seaweeds. It can reproduce asexually, and an entire individual can be regenerated from its blood vessels alone.
Focusing on genes involved in various human diseases – affecting things such as heart and eye development, pregnancy and cancer – they found homologous genes for each in Botryllus, far more matches than in any of a dozen other invertebrates commonly used in research.
An additional investigation of blood-related genes revealed that Botryllus was probably the first invertebrate to have vasculature in the same context of the human circulatory system, with blood cells travelling through blood vessels.
“The whole body can regenerate from the vasculature alone: the heart, digestive system, sophisticated tissues,” said Ayelet Voskoboynik, the lead author on the study.
“And it can do this relatively fast, probably using stem cells. Now that we have the genome, we can try to understand the mechanism behind it,” Voskoboynik said.
Jawanda Mast helps her daughter Rachel, 14, write thank you notes at their home in Olathe, Kansas. Rachel has Down syndrome.
In the 14 years since her daughter, Rachel, was born with Down syndrome, Jawanda Mast has always been clear that she’d change the condition if she could.
“I couldn’t love her more, but I would give almost anything to take away that extra chromosome,” the Olathe, Kansas, mom wrote on her blog. “While I may know she’s perfect, the world doesn’t.”
But when Massachusetts scientists announced recently that they’ve found a way to silence the chromosome that causes trisomy 21, also known as Down syndrome, it rocked Mast – and the rest of the disability community.
“It’s so hard to imagine you could actually do that,” Mast told NBC News. “Yes, I would take away the challenges, I would take away the health risks. But now I also stop and say, ‘Oh my goodness, how would that impact the rest of her?’”
Hailed as a “cure in a Petri dish,” the research by scientists at the University of Massachusetts Medical School is the first to find that it may be possible to switch off the genetic material responsible for the condition that causes cognitive delays, heart defects and shortened lifespans.
The development is expected to help create new treatments for problems caused by Down syndrome — but it also raises the prospect of eliminating the condition entirely.
Since it became public last month, the breakthrough has sparked a firestorm of reaction among parents, advocates, ethicists and people with the condition, said Dr. Brian Skotko, a medical geneticist and co-director of the Down Syndrome Program at Massachusetts General Hospital.
“This research really launches a million questions,” Skotko said.
Chuck France / Getty Images for NBC News
At left, Rachel Mast, 14, is training with other students at her middle school to become student ambassadors to incoming sixth-graders.
On one hand, almost everyone agrees there’s a need for treatments to help the 250,000 people in the U.S. living with Down syndrome, including the nearly 7,000 babies born with it each year.
On the other hand, it’s unclear what costs there may be to shutting down the mechanism that creates people who offer lessons in patience, kindness — and what it means to be human.
“If Down syndrome were completely cured, the world would lose something from the absence of that culture,” said Skotko, who has a sister with the condition. “There is something positive that people with Down syndrome contribute to the world.”
Brian Long of Boulder, Colo., is the father a 19-year-old son with Down syndrome. He welcomes the research, which could lead to treatments to boost Connor’s intellectual abilities and speaking skills and prevent disease. But he also wonders how tinkering with chromosomes could alter the essence of his son.
“So much of Down syndrome does impact the personality and character of the person,” said Long, 54. “In Connor’s example, we’ve known him for 19 years. We don’t want a wholesale change.”
Advocates like Julie Cevallos, vice president of marketing for the National Down Syndrome Society, emphasize that the research is still early.
“When you go as far as a ‘cure,’ that’s when folks step back and go: ‘We’re not looking for a cure. We’re looking to help and support people with Down syndrome live healthy and productive lives,’” said Cevallos, mother of a 5-year-old with the condition.
David Egan, a 35-year-old Vienna, Va., man with Down syndrome, said he applauds the progress in part because it might help with some of the social stigma that comes with the disorder. He has friends who’ve been made fun of because of their disability, who have a hard time coping with the condition.
Stone uncovered last night on the Ness of Brodgar in Orkney, Scotland
Found at base of Neolithic cathedral’s south-west internal corner at site
Excavations began in 2003 and buildings and houses have been found
Archaeologists have found an astonishing piece of Neolithic artwork that was buried for 4,500 years.
The stone creation – which is decorated on both sides and has been described as one of the ‘finest ever’ to be found in Britain – was uncovered last night on the Ness of Brodgar in Orkney, Scotland.
It was found at the base of the south-west internal corner of the Neolithic ‘cathedral’ at the site, which covers 2.5 hectares and is believed to have been occupied from as early as 3,500BC.
Impressive: The stone art work – which is decorated on both sides and has been described as one of the ‘finest ever’ to be found in Britain – was uncovered last night on the Ness of Brodgar in Orkney, Scotland
Ancient: The other side of the stone, whch was found at the base of the south-west internal corner of the Neolithic ‘cathedral’ at the site, which covers 2.5 hectares and is believed to have been occupied from 3500BC
Neolithic man built a vast temple complex at the Ness of Brodgar, with some parts constructed more than 5,000 years ago, even before the Ancient Egyptians had started building the pyramids.
Excavations began in 2003 at the site, which has provided evidence of housing, decorated stone slabs, a massive stone wall with foundations, and a large building described as a Neolithic cathedral
Once protected by two giant walls, each more than 330ft long and 13ft high, the complex at the Ness of Brodgar contained more than a dozen large temples, with one measuring almost 270 sq ft.
They were linked to outhouses and kitchens by carefully constructed stone pavements. The bones of sacrificed cattle, elegantly made pottery and pieces of painted ceramics lie scattered there.